The discovery of MaRNAV-7, a Matryoshka RNA virus, in a common whitethroat bird co-infected with avian malaria parasites, marks a significant advancement in understanding avian virology. This is the first identification of MaRNAV in an African-European haemosporidian parasite-infected common whitethroat. The common whitethroat was primarily infected with *Leucocytozoon* sp. lRECOB3, a parasite reported across Europe, Africa, and Asia. This finding underscores the complex interactions between viruses and parasites within avian hosts and opens new avenues for research into host-parasite-vector relationships.

Matryoshka RNA viruses, or marnav, are increasingly recognized for their role in influencing host immunological responses, parasite virulence, parasitemia, and transmission rates. These viruses, found in conjunction with haemosporidian parasites in birds, are the subject of ongoing investigation. Understanding the relationship between these viruses and their hosts is crucial for determining their impact on the overall health and ecology of avian populations.

Haemosporidian parasites, including genera like *Plasmodium*, *Leucocytozoon*, and *Haemoproteus*, are widespread in avian populations. These parasites, responsible for diseases akin to malaria in birds, have complex life cycles involving both avian hosts and insect vectors. Recent research has focused on the viruses that infect these parasites, potentially influencing the course of infection and disease severity. The discovery of MaRNAV-7 adds another layer of complexity to this already intricate system.

The homology-based search for MaRNAV-7 revealed transcripts encoding for RdRp with 38–81% similarity to previous MaRNAV RdRps, and also additional genes encoding the hypothetical protein from MaRNAV-2. This suggests an evolutionary relationship between MaRNAV-7 and other known MaRNAV viruses, particularly MaRNAV-2 found in Australian birds infected with *Leucocytozoon* parasites.

The available data suggest that MaRNAV-7 infects a *Leucocytozoon* sp.(lineage lRECOB3) because:

  1. It is closely related to MaRNAV-2 found in Australian birds infected with *Leucocytozoon* parasites.
  2. The *Leucocytozoon* parasitemia was significantly higher than that of the other haemosporidians in the host.
  3. Transcriptomes BLASTn resulted in hits to *Leucocytozoon* sequences, but no hits for *Haemoproteus* or *Plasmodium*.
  4. A second fragment (hypothetical protein) was also found in the sample, similar to other MaRNAV *Leucocytozoon*-infected birds.

The common whitethroat in this study was primarily infected with *Leucocytozoon* sp. lRECOB3, which has been reported in Europe, Africa, and Asia across 13 countries and 26 bird species from 12 bird families (MalAvi, accessed October 22, 2024). Though,this is only one record of MaRNAV in Europe,and its prevalence remains unknown.

The diagnosis of MaRNAV in avian hosts infected with haemosporidian parasites is challenging due to the high mutation rates of RNA viruses, which can reduce the efficiency of conventional PCR protocols unless specific-MaRNAV strain primers are used. Future research targeting bird species known to be infected with lRECOB3 may help to understand the prevalence of MaRNAV-7 in wildlife.

The recent finding of the same virus in different bird species and even orders suggests the possibility of vector involvement in the infection process.The study suggests that the transmission of the virus from one parasite to another would happen in the vector, which would then transmit the associated parasites-virus to different bird species in the same locality. This hypothesis is supported by the fact that vectors are in contact with a high diversity of hosts, bacteria, and viruses, and virus distribution could be correlated with vector occurrence.

One key area of interest is the tissue stages of these parasites. While *Plasmodium* species are known for their phanerozoites, *Leucocytozoon* and *Haemoproteus* exhibit megalomeronts and meronts. The specificity of marnav to species with certain tissue stages remains a question. As noted, It is possible that MaRNAV might be specific to species with certain tissue stages. Though, the life cycle is not completely known for most of the avian haemosporidian species.

The presence of hypnozoites, a dormant stage in the life cycle of some *Plasmodium* species, adds another layer of complexity. *Plasmodium cynomolgi* in monkeys and *P. berghei* in rodents are examples of species that develop hypnozoites. while SRA data were screened for MaRNAV in these species, the viruses were not found. It’s critically important to note that most of the records came from experiments and cultures, which might impact the virus prevalence. Therefore, investigating natural infections of *Plasmodium* species that develop hypnozoites in different hosts is essential to clarify this question.

Further research is needed to fully understand the diversity, prevalence, and impact of MaRNAV. Studies targeting MaRNAV and their relationship to haemosporidian parasite life cycles are essential. As emphasized, Further studies targeting MaRNAV and how they are related to haemosporidian parasites life cycle are essential to understand not only their diversity and prevalence, but also their effect on host immunological response modulation, parasites virulence, parasitemia and transmission rates and host-parasite-vectors relationships.